Yingchun Ni is an associate in the San Diego office of Wilson Sonsini Goodrich & Rosati. His practice focuses on patent portfolio development and patent strategy counseling for clients in the biotechnology and pharmaceutical industries. He advises clients on matters related to patent preparation and prosecution, patent strategy, patent portfolio management, freedom-to-operate, due diligence, patentability, and non-infringement and invalidity analysis. Yingchun has represented clients of early-stage start-ups, clinical-stage companies, and venture capital firms in connection with financings, collaboration and licensing agreements, public offerings, and mergers and acquisitions.
Prior to his legal career, Yingchun obtained his Ph.D. in Neuroscience from the University of California, Riverside, where he studied neurotransmitter release. After graduate school, he conducted postdoctoral studies at the National Institutes of Health and Boston Children’s Hospital and his postdoctoral research focused on neural signaling and neural circuits.
Yingchun is a native speaker of Mandarin Chinese.
Yingchun Ni is an associate in the San Diego office of Wilson Sonsini Goodrich & Rosati. His practice focuses on patent portfolio development and patent strategy counseling for clients in the biotechnology and pharmaceutical industries. He advises clients on matters related to patent preparation and prosecution, patent strategy, patent portfolio management, freedom-to-operate, due diligence, patentability, and non-infringement and invalidity analysis. Yingchun has represented clients of early-stage start-ups, clinical-stage companies, and venture capital firms in connection with financings, collaboration and licensing agreements, public offerings, and mergers and acquisitions.
Prior to his legal career, Yingchun obtained his Ph.D. in Neuroscience from the University of California, Riverside, where he studied neurotransmitter release. After graduate school, he conducted postdoctoral studies at the National Institutes of Health and Boston Children’s Hospital and his postdoctoral research focused on neural signaling and neural circuits.
Yingchun is a native speaker of Mandarin Chinese.
- J.D., University of Michigan Law School, 2019
Dean’s Scholarship; Managing Articles Editor, Michigan Technology Law Review
- Ph.D., Neuroscience, University of California, Riverside, 2006
- B.S., Biophysics, Fudan University, 2000
- State Bar of Arizona
- State Bar of California
- U.S. Patent and Trademark Office
- J.D., University of Michigan Law School, 2019
Dean’s Scholarship; Managing Articles Editor, Michigan Technology Law Review
- Ph.D., Neuroscience, University of California, Riverside, 2006
- B.S., Biophysics, Fudan University, 2000
- State Bar of Arizona
- State Bar of California
- U.S. Patent and Trademark Office
Select Publications
- Co-author, “Characterization of long descending premotor propriospinal neurons in the spinal cord,” 34 (28) J Neurosci. 9404-9417, 2014
- Co-author, “Non-synaptic communication through ATP release from volume-activated anion channels in axons,” 3(142) Sci. Signal. ra73, 2010
- Co-author, “Dual regulation of Ca2+-dependent glutamate release from astrocytes: vesicular glutamate transporters and cytosolic glutamate levels,” 57(12) Glia 1296-1305, 2009
- Co-author, “Ca2+ entry through TRPC1 channels contributes to intracellular Ca2+ dynamics and consequent glutamate release from rat astrocytes,” 56(8) Glia 821-835, 2008
- Co-author, “Vesicular release of glutamate mediates bidirectional signaling between astrocytes and neurons,” 103(4) J Neurochem. 1273-1284, 2007
- Co-author, “Vesicular glutamate transporter-dependent glutamate release from astrocytes,” 24(11) J Neurosci. 2633- 2642, 2004
Select Publications
- Co-author, “Characterization of long descending premotor propriospinal neurons in the spinal cord,” 34 (28) J Neurosci. 9404-9417, 2014
- Co-author, “Non-synaptic communication through ATP release from volume-activated anion channels in axons,” 3(142) Sci. Signal. ra73, 2010
- Co-author, “Dual regulation of Ca2+-dependent glutamate release from astrocytes: vesicular glutamate transporters and cytosolic glutamate levels,” 57(12) Glia 1296-1305, 2009
- Co-author, “Ca2+ entry through TRPC1 channels contributes to intracellular Ca2+ dynamics and consequent glutamate release from rat astrocytes,” 56(8) Glia 821-835, 2008
- Co-author, “Vesicular release of glutamate mediates bidirectional signaling between astrocytes and neurons,” 103(4) J Neurochem. 1273-1284, 2007
- Co-author, “Vesicular glutamate transporter-dependent glutamate release from astrocytes,” 24(11) J Neurosci. 2633- 2642, 2004
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